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Table 1 Dyadicity and heterophilicity analysis results of the bladder cancer gene interaction network

From: Functional dyadicity and heterophilicity of gene-gene interactions in statistical epistasis networks

Gene Ontology terms n 1 m 11 m 10 \(\overline {m}_{11}\) \(\overline {m}_{10}\) D H p D p H
Nucleoplasm 30 9 38 4.422 47.265 2.035 0.804 0.042 0.954
Identical protein binding 27 1 56 3.568 43.362 0.280 1.291 0.977 0.026
Response to estrogen stimulus 10 1 29 0.457 17.788 2.186 1.630 0.401 0.015
Transcription, DNA-dependent 9 2 25 0.366 16.101 5.466 1.553 0.062 0.036
RNA biosynthetic process 9 2 25 0.366 16.101 5.466 1.553 0.064 0.036
Steroid binding 8 2 12 0.285 14.393 7.027 0.834 0.042 0.776
Axon 6 2 7 0.152 10.917 13.118 0.641 0.013 0.952
Negative regulation of DNA binding 5 2 11 0.102 9.148 19.676 1.202 0.006 0.301
Regulation of phagocytosis 3 1 7 0.030 5.550 32.794 1.261 0.045 0.260
Nucleotide-excision repair, DNA gap filling 3 0 12 0.030 5.550 0 2.162 1 0.037
Regulation of sterol-transport 3 1 5 0.030 5.550 32.794 0.901 0.047 0.616
Regulation of cholesterol transport 3 1 5 0.030 5.550 32.794 0.901 0.048 0.618
  1. A 100,000-fold permutation testing was used to estimate the significance levels of the calculated D and H, and the p-values less than the threshold 0.05 were noted in bold font